Training Faculty Profiles
Our Training Faculty are 95 highly distinguished scientists who have made fundamental discoveries in all areas of neuroscience from molecules to cognition. They have international reputations for excellence in both research and teaching, and are actively involved in training of Program students through direct mentorship, thesis committee participation, instruction of program courses, and/or participation in program leadership. Browse our faculty by sorting through one of the methods below:
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We are interested in understanding the mechanisms by which neuroinflammation elicits synaptic and neuronal injury in chronic and acute models of neurological disease. Our foot in the door has been the study of the cyclooxygenase-2 (COX-2) pathway and its downstream prostaglandin receptor signaling pathways, which we have discovered function in very important ways in modulating the inflammatory response in brain in models of Alzheimer's disease (AD), amyotrophic lateral sclerosis (ALS), Parkinson's disease (PD), and stroke. Thus this pathway functions across a broad spectrum of neurodegenerative diseases, and may potentially modulate inflammatory responses and neuronal injury via conserved cellular and molecular mechanisms. We use genetic and pharmacologic strategies as well as in vitro culture approaches to define COX-2/prostaglandin receptor mediated mechanisms of action in eliciting synaptic and neuronal injury in these models of human neurological disease. Our long-term goal is to (1) further understand how neuroinflammatory processes injure synapses and neurons and disrupt circuits, (2) define the contribution of the COX-2/prostaglandin signaling pathways in this process, and (3) develop therapeutic strategies targeting prostaglandin G-protein coupled receptors in human neurological diseases.
http://neurology.stanford.edu/labs/andreassonlab/
http://neurology.stanford.edu/labs/andreassonlab/
Visual processing in neural circuits of the retina, studied using multielectrode extracellular array recording, intracellular recording, two-photon imaging, and computational modeling.
http://med.stanford.edu/profiles/neuroscience/faculty/Stephen_Baccus
http://med.stanford.edu/profiles/neuroscience/faculty/Stephen_Baccus
Our lab is interested in the neuronal-glial interactions that underlie the development, function, and regeneration of the mammalian central nervous system.
http://med.stanford.edu/profiles/neuroscience/faculty/Ben_Barres
http://med.stanford.edu/profiles/neuroscience/faculty/Ben_Barres
Cancer and stem cells can be viewed as two sides of the same coin. Regenerative medicine entails increasing the plasticity of cell fate. However, this increase in cellular plasticity also increases the chances of cancer. For example, to produce induced pluripotent stem cells (iPS), two of the four Yamanaka proteins are onco-proteins (c-Myc and Klf4). To create cells for regeneration by dedifferentiation, the activity of two tumor suppressors (RB and p61/p19) is transiently inhibited. Thus, in order to enlist cells for tissue regeneration, the chances for cancer are increased and must be carefully monitored. We have shown that by non-invasive imaging by bioluminescence (BLI) we can distinguish in a dynamic and quantitative manner whether injected cells are functioning as expected for regeneration or have gone awry as in cancer. For example, the development of additional radiological methods for tracking single human cells delivered in vivo (e.g. using nanoparticles) has clear potential application in distinguishing tumorigenic or normal regenerative behavior. For regenerative medicine to succeed, cancer must be prevented, detected and controlled. Radiologic tools and technologies are invaluable to both regeneration and cancer and more such methods need to be developed and actively employed. This is a strong multidisciplinary focus of my laboratory.
http://med.stanford.edu/profiles/neuroscience/faculty/Helen_Blau
http://med.stanford.edu/profiles/neuroscience/faculty/Helen_Blau
Our group has two synergistic goals: to understand how brains work, which will enable us to replace damaged neural tissue, and to build computers that work like brains, which will enable us to increase computational power a million-fold. To these ends, we model brains using an approach far more efficient than software simulation: we emulate the flow of ions directly with the flow of electrons---don't worry, on the outside, it looks just like software.
http://brainsinsilicon.stanford.edu
http://brainsinsilicon.stanford.edu
Our lab studies the molecular basis of aging, with an emphasis on the role of the nervous system in longevity. We use worms, fish, and mice to discover novel genes that regulate aging and to study the importance of these genes in the nervous system. We are particularly interested in the role of longevity genes in preserving the adult neural stem cell pool and in preventing the decline in cognitive behaviors during aging. Our lab also explores if specific brain regions secrete factors that control the overall aging process.
http://www.stanford.edu/group/brunet/
http://www.stanford.edu/group/brunet/
Axel Brunger's goal is to understand the molecular mechanism of synaptic neurotransmission. He is particularly interested in the structure, function, and dynamics of key players in the synaptic vesicle fusion machinery. His lab is also working on the mechanism of action of clostridial neurotoxins that target this machinery. Other projects include the ATPases of the AAA family that are involved in protein complex disassembly and degradation. A molecular understanding of these complex protein machineries may ultimately lead to new therapeutics to treat human diseases.
http://atb.slac.stanford.edu
http://atb.slac.stanford.edu
Mechanisms of epilepsy; circuitry of temporal lobe structures.
http://med.stanford.edu/profiles/neuroscience/faculty/Paul_Buckmaster
http://med.stanford.edu/profiles/neuroscience/faculty/Paul_Buckmaster
Our lab focuses on how inflammatory responses after brain injury affect neurological recovery. In the United States, there are 4 million people currently living with the effects of stroke, and another 4.3 million living with the effects of traumatic brain injury. Of the people who have had a stroke, many are disabled to the degree that they cannot work, and a significant proportion are unable to walk, feed themselves, or communicate with their families the way they could prior to their stroke. Despite this very high number of people who are suffering, there is a large knowledge gap regarding the mechanisms by which neurological recovery occurs, and not a single FDA-approved therapy available to help people recover. There is reason to think that such a therapy might be obtainable – we know that some people, especially younger ones, experience significant recovery after stroke. Animal studies, almost entirely done in young animals, also demonstrate significant recovery after neurological injury. Our goal is thus to better understand the mechanisms that contribute to recovery in the young, and how they are influenced by inflammatory responses. Once we understand this, we hope to be able to develop new therapies to help people’s brains repair themselves.
Current projects in the lab:
TGF-beta signaling after brain injury. To understand the role of TGF-beta signaling after brain injury, we use mouse models to manipulate and image TGF-beta signaling after stroke, viral vectors to influence TGF-beta signaling in neural progenitor cells, and small molecule therapies in a time-restricted fashion. We measure the effects on functional recovery from brain injury, the cellular and molecular immune response, and cell-specific signaling pathways.
The effect of small molecule neurotrophin agonists on functional recovery. In collaboration with the Longo lab, which has developed these compounds, we are testing whether small molecule compounds that mimic NGF and BDNF can be used to improve recovery and stimulate regenerative responses after brain injury.
Imaging and manipulating regenerative responses after brain injury. Constructing novel mouse models to allow for real-time imaging and manipulation of neurogenesis and oligodendrogenesis in mice as they recover from brain injury.
Peripheral immune responses and brain edema after stroke. In collaboration with researchers at the Stanford Stroke Center we plan to evaluate serum samples from patients with stroke. Our goal is to understand the peripheral immune mechanisms that correlate with the development of brain edema, or swelling, and determine if there are ways to predict which patients may require more aggressive treatment for their strokes.
http://med.stanford.edu/profiles/neuroscience/faculty/Marion_Buckwalter
http://med.stanford.edu/profiles/neuroscience/faculty/Marion_Buckwalter
We are particularly interested in synaptic signaling mechanisms that govern the strength of synaptic transmission and circuit properties, as well as their impact on animals' ability to learn and memorize.
http://chen-lab.stanford.edu
http://chen-lab.stanford.edu
Our long-term goal is to understand how sensory information and physiological state integrate to drive behaviors and influence decisions. We are now focusing on interoception, which is the sense of the physiological condition of the body. This includes our abilities to feel hungry or satiated, to sense heightened blood pressure and heart rate during stress, and to discriminate different types of pain. In our lab, we combine genetics-based brain circuit manipulation with in vivo electrophysiological and optical recording of neuronal ensembles to dissect the coding logic underlying interoception.
http://www.stanford.edu/group/chen_lab/
http://www.stanford.edu/group/chen_lab/
My laboratory studies childhood brain tumors with a particular focus on medulloblastoma, the most common malignant brain tumor in children. We utilize computational and cell biological approaches to understand the molecular and cellular basis of this disease. Current projects include 1) leveraging expression of neurotransmitter receptors in brain tumors and other cancers for diagnostic and therapeutic purposes, 2) investigating the role of ATP-dependent RNA helicases in neural development and disease and 3) developing a functional annotation of the medulloblastoma genome by merging whole-genome RNAi with high-throughput chemical biology and chemical genomic screens.
http://med.stanford.edu/profiles/neurology/researcher/Yoon-Jae_Cho/
http://med.stanford.edu/profiles/neurology/researcher/Yoon-Jae_Cho/
Genetic and molecular mechanisms controlling the development of precise patterns of neuronal connections in the central nervous system. Functional dissection of neuronal circuits controlling visual behaviors in the fruit fly.
http://med.stanford.edu/profiles/neuroscience/faculty/Thomas_Clandinin
http://med.stanford.edu/profiles/neuroscience/faculty/Thomas_Clandinin
Structural organization and function of peripheral and central neural pathways that underlie directed manual behavior in the nonhuman primate. Capacity of these neural pathways to compensate/adapt following specific sensory manipulations.
http://med.stanford.edu/profiles/neuroscience/faculty/Corinna_Darian-Smith
http://med.stanford.edu/profiles/neuroscience/faculty/Corinna_Darian-Smith
We use a combination of optogenetic, pharmacological, molecular and behavioral methods to map and manipulate the neuronal circuits controlling sleep, arousal and hyperarousal (i.e. stress and addiction).
http://med.stanford.edu/profiles/neuroscience/faculty/Luis_de Lecea
http://med.stanford.edu/profiles/neuroscience/faculty/Luis_de Lecea
Neural stem cells, neuroengineering, adaptive plasticity, electrophysiology, two-photon imaging, animal behavior, computational modeling, neuropsychiatry, developing noninvasive technologies for focal brain stimulation.
http://www.stanford.edu/group/dlab/
http://www.stanford.edu/group/dlab/
Although stress generally has a “bad” reputation, a short-term stress is response is nature's fundamental protective mechanism without which neither predator nor prey could survive. We are interested in identifying biological mechanisms that mediate and differentiate the recently appreciated immunoenhancing effects of short-term stress (eustress) from the well-known immunosuppressive effects of long-term stress (distress). We examine stress effects on the neuroendocrine system, and on leukocyte trafficking, innate/adaptive immunity, and cytokine gene/protein expression using models of skin immunity, surgery, and cancer.
http://med.stanford.edu/profiles/neuroscience/faculty/Firdaus_Dhabhar
http://med.stanford.edu/profiles/neuroscience/faculty/Firdaus_Dhabhar
The interplay between motor cortex, sensory cortex, thalamus and basal ganglia is essential for neural computations involved in generating voluntary movements. Our goal is to dissect the functional organization of motor circuits, particularly cortico-thalamo-basal ganglia networks, using electrophysiology, 2-photon microscopy, optogenetics, and genetic tools. The long-term scientific goal of the lab is to construct functional circuit diagrams and establish causal relationships between activity in specific groups of neurons, circuit function, animal motor behavior and motor learning, and thereby to decipher how the basal ganglia process information and guide motor behavior. We will achieve this by investigating the synaptic organization and function that involve the cortex, thalamus and basal ganglia at the molecular, cellular and circuit level. Currently, we are focusing on several questions:
How are excitatory inputs integrated in the striatum?
How do feed-forward and recurrent local inhibitions balance the excitation in the striatum?
How are functional maps modulated in motor behavior and motor learning?
Our goal is to bridge the gap between molecular or cellular events and the circuit mechanisms that underlie motor behavior. In addition, we aim to further help construct the details of psychomotor disorder ‘circuit diagrams,’ such as the pathophysiological changes in Parkinson’s disease.
We use a combination of molecular biology, microscopy, electrophysiology and stem cell biology to study the biological basis of autism. We are also interested in calcium channels and calcium signaling. Finally we are interested in developing new techniques for studying the brain.
http://www.stanford.edu/group/dolmetschlab/
http://www.stanford.edu/group/dolmetschlab/
The overarching aim of the Etkin lab is to understand the neural basis of emotional disorders and their treatment, and to leverage this knowledge to develop novel treatment interventions. Clinical experience and data suggest that abnormalities in the regulation of emotional processing, in particular through mechanisms operating outside of awareness (i.e. implicit) are central to a range of psychopathology. Thus, our investigation of psychopathology and treatment is organized around affective neuroscience of emotion regulation.
Implicit emotion regulation: A successful affective neuroscience approach to psychopathology and treatment requires understanding the basic mechanisms involved in emotion regulation. Although our initial work thus far has yielded important insights, we are far from a thorough understanding of how emotion is implicitly regulated. Ongoing work in the lab is focused on understanding the factors which govern implicit emotion regulation, the relationship between implicit and explicit regulation, and whether there are ways to improve implicit emotion regulation through training.
Neural basis of psychopathology: Our recent work suggests that a deficit in implicit emotion regulation may be a core feature of anxiety, which is evident in patients with generalized anxiety disorder (GAD), including in the context of major depressive disorder (MDD). We are also currently examining how patients with different, but related, conditions, such as post-traumatic stress disorder (PTSD) and chronic pain implicitly regulate emotion and how this reflects common versus disorder-specific neural signatures. In taking a life-span perspective on implicit emotion regulation, we are also currently studying older healthy subjects and those with geriatric anxiety or depression.
Neural mechanisms of treatment: Very little is known about the mechanisms of action of existing treatments in psychiatry, which is particularly true of psychotherapy, despite the importance of this clinical tool. An important goal of the lab is understanding the neural processes involved in treatment for anxiety or depression, in terms of (a) which domains of neural/mental functions are involved, (b) how different approaches yield their effects, (c) how individual differences in capacities like emotion regulation underlie differential outcome, and (d) how the mechanisms of change with medication relate to and interact with those involved in psychotherapy. We are currently NIH-funded to study the emotion regulation mechanisms underlying exposure therapy for PTSD.
Neural circuits subserving emotion: An element integral to the studies above is a delineation of the neural circuits that underlie emotion processing. We have, for example, demonstrated that the major amygdalar subregions in humans have distinct patterns of resting-state functional connectivity, which are perturbed in GAD. Ongoing work in the lab is focused on extending this mapping of circuitry important for emotion, using functional connectivity, in both healthy subjects and patients with mood or anxiety disorders.
http://etkinlab.stanford.edu/
http://etkinlab.stanford.edu/
Reproduction is the most powerful selective force in evolution and we focus on how important information about sex changes the nervous system. We study how social information is transduced into cellular and molecular changes using a range of techniques from behavioral observation to molecular analyses. Since we have shown that certain brain cells containing gonodotropin releasing hormone respond to changes in social status by changing size and connectivity, we are now examining the mechanisms including the role(s) of micro RNAs as well as epigenetic processes such as methylation of regulatory genes.
http://www.stanford.edu/group/fernaldlab/index.shtml
http://www.stanford.edu/group/fernaldlab/index.shtml
Our lab works on theoretical neuroscience, with the fundamental goal of understanding how networks of neurons and synapses cooperate across multiple scales of space and time to mediate important brain functions, like sensory perception, motor control, and memory. To achieve this goal, we employ and extend tools from disciplines like statistical mechanics, dynamical systems theory, machine learning, information theory, control theory, and high-dimensional statistics, as well as collaborate with experimental neuroscience laboratories collecting physiological data from a range of model organisms. Some topics of interest include: how birds learn to sing, spatial memory in the rodent hippocampus, attention and motor control in macaques, memory properties of complex synapses, dynamics of plasticity in recurrent networks, signal propagation in neural circuits, the emergence of categorization in multi-layered networks, and the statistical mechanics of high dimensional data analysis.
http://www.stanford.edu/dept/app-physics/cgi-bin/person/surya-gangulijanuary-2012/
http://www.stanford.edu/dept/app-physics/cgi-bin/person/surya-gangulijanuary-2012/
Our laboratory is interested in the molecular mechanisms that regulate the dynamic assembly and function of vertebrate synapses. Current efforts are oriented towards understanding how genetic lesions and environmental insults alter synaptic plasticity mechanisms and neuronal network function in neurodevelopmental disorders such as Down syndrome and autism. We also have an active translational program that takes insights from experimental neuroscience and develops rational clinically relevant pharmacotherapies. The first for treating cognitive impairment in Down syndrome will enter the clinic spring of 2012.
http://med.stanford.edu/profiles/neuroscience/faculty/Craig_Garner
http://med.stanford.edu/profiles/neuroscience/faculty/Craig_Garner
The lab has a special emphasis in problems without advocates - from under-served disorders in women (such as trichotillomania), to animal welfare. Similarly, the lab especially welcomes students from under-represented backgrounds. I myself am a first-generation student, and I take great pride in a long track record of successfully graduating students from under-represented backgrounds, including generation, minority, LGBTQ, and female students, veterans and students with disabilities. The lab uses an integrated interdisciplinary approach, best described as developmental neuroethology, to address issues in human and animal well-being. The lab has a particular focus on two closely related issues: 1) Developing methods and underlying psychobiological principles to predict and prevent abnormal behavior (in animals) and mental disorder (in humans). 2) Identifying the general reasons why animal models often fail to predict human outcomes, and providing solutions to improve the efficacy and well-being of animal models. Both these issues reflect the interface between animal-based medical research, and animal well-being. The medical research community has long recognized that "good well-being is good science" – the lab's work is directed at exploring this interface, while providing tangible deliverables for the well-being of human patients and research animals.
For instance, current projects in the lab include: (on the animal well-being side) optimal design and the impacts of nesting enrichments on the behavior, physiology, and well-being of laboratory mice; and (on the human health side) development of predictive biomarkers and preventative dietary interventions in a mouse model of trichotillomania (compulsive hair pulling). The lab also works collaboratively on farm-animal and zoo-animal well-being issues with colleagues around the world.
med.stanford.edu/compmed/research/garner_lab.html
http://med.stanford.edu/profiles/compmed/faculty/Joseph_Garner
http://med.stanford.edu/profiles/compmed/faculty/Joseph_Garner
Cellular and molecular basis for neuronal and astrocyte vulnerability to ischemia; roles of chaperones, roles of microRNAs, inflammation and mitochondria in cell death, modeling death pathways.
http://med.stanford.edu/profiles/neuroscience/faculty/Rona_Giffard
http://med.stanford.edu/profiles/neuroscience/faculty/Rona_Giffard
My laboratory studies the cellular and molecular mechanisms underlying the organization of cortical circuits important for spatial navigation and memory. We are particularly focused on medial entorhinal cortex, where many neurons fire in spatially specific patterns and thus offer a measurable output for molecular manipulations. We combine electrophysiology, genetic approaches and behavioral paradigms to unravel the mechanisms and behavioral relevance of non-sensory cortical organization.
Our first line of reached is focused on determining the cellular and molecular components crucial to the neural representation of external space by functionally defined cell types in entorhinal cortex (grid, border and head direction cells). We plan to use specific targeting of ion channels, combined with in vivo tetrode recordings, to determine how channel dynamics influence the neural representation of space in the behaving animal. A second, parallel line of research, utilizes a combination of in vivo and in vitro methods to further parse out ionic expression patterns in entorhinal cortices and determine how gradients in ion channels develop. Ultimately, our work aims to understand the ontogenesis and relevance of medial entorhinal cortical topography in spatial memory and navigation.
We investigate the mechanisms of human neurodegenerative diseases, including Alzheimer's disease, Parkinson's disease, and ALS. We don't limit ourselves to one model system or experimental approach. We start with yeast, perform genetic and chemical screens, and then move to other model systems (e.g. mammalian tissue culture, mouse, fly) and even work with human patient samples (tissue sections, patient-derived cells, including iPS cells) and next generation sequencing approaches.
Development of novel methods for imaging of brain function using MRI
http://rsl.stanford.edu/glover
http://rsl.stanford.edu/glover
Cellular and molecular basis of sensory mechano- and thermotransduction. We study sensation at the molecular, cellular and organismal levels, leveraging the complete wiring diagram of the C. elegans nervous system, advanced tools in classical and molecular genetics, electron microscopy, and in vivo electrophysiology.
http://wormsense.stanford.edu/
http://wormsense.stanford.edu/
Neural foundations of affective disorders; psychobiology of depression and anxiety; risk for depression in children and adolescents.
http://mood.stanford.edu
http://mood.stanford.edu
fMRI, computational and behavioral studies of visual perception.
http://vpnl.stanford.edu
http://vpnl.stanford.edu
Neural and autonomic bases of emotion and emotion regulation: basic processes (emphasizing relations among behavior, physiology, and subjective experience); personality correlates; health implications, with particular emphasis on social anxiety disorder.
http://med.stanford.edu/profiles/neuroscience/faculty/James_Gross
http://med.stanford.edu/profiles/neuroscience/faculty/James_Gross
Neurobiology of sleep, circadian rhythms, regulation of body temperature, mammalian hibernation, and human exercise physiology. Dr. Heller is co-director of the Center for Sleep and Circadian Neurobiology. The Center fosters multidisciplinary approaches and collaborations that will help us understand the neural mechanisms controlling arousal states and arousal state transitions, the function of sleep, and the neural mechanisms of circadian rhythms. Research on human exercise physiology focuses on the effects of body temperature on physical conditioning and performance.
http://med.stanford.edu/profiles/neuroscience/faculty/Craig_Heller
http://med.stanford.edu/profiles/neuroscience/faculty/Craig_Heller
The main interest of my lab is to understand how the properties of neocortical neurons and the circuits they form give rise to cortical activity and function. We study neuronal activity in slices and in vivo in awake behaving animals.
http://med.stanford.edu/profiles/neuroscience/faculty/Shaul_Hestrin
http://med.stanford.edu/profiles/neuroscience/faculty/Shaul_Hestrin
The balance between the removal and production of ROS determines the reduction and oxidation (redox) status in tissues, cells, and subcellular compartments. Under pathological conditions, such as infection or inflammation, or exposure to external stressors, such as toxic chemicals or irradiation, the production of ROS exceeds the antioxidant capacity and tips the redox balance to causing cell death and tissue dysfunction. To determine how changes in redox balance affect tissue maintenance and repair, we use tissue culture cells and mouse models with altered antioxidant capacity and subject these experimental models to various forms of oxidative stress. One of the current research focuses is on the effects of redox balance on hippocampal neurogenesis
http://med.stanford.edu/profiles/neuroscience/faculty/Ting-Ting_Huang
http://med.stanford.edu/profiles/neuroscience/faculty/Ting-Ting_Huang
Neurobiology of thalamocortical oscillatory activities in epilepsy and sleep. Mechanisms of hyperexcitability, neuronal hypersynchrony, and relevant antiepileptic drug actions. Development of neocortical and thalamic networks. Computational models of realistic neural networks.
http://huguenardlab.stanford.edu
http://huguenardlab.stanford.edu
We are using genetics and genomics to identify specific genes and mutations that underlie new morphological, physiological, and behavioral traits during vertebrate evolution, including brain expansion and other unique morphological traits in humans. Approaches used include genome-wide linkage mapping of recent evolutionary change in threespine stickleback fish; genome-wide analysis of likely functional changes in lizards, whales, chimps, and humans; and detailed experimental tests of likely functional changes using transgenic, knock-out, and knock-in animals.
http://kingsley.stanford.edu
http://kingsley.stanford.edu
Systems, circuit and synaptic mechanisms of spatial attention, studied in developing and adult owls and chickens, using behavioral, systems, in vitro slice, extracellular and patchclamp recording, pharmacology, and molecular techniques.
http://med.stanford.edu/profiles/neuroscience/faculty/Eric_Knudsen
http://med.stanford.edu/profiles/neuroscience/faculty/Eric_Knudsen
Role of biogenic amines in modulating emotional experience. Neural substrates of incentive processing, with implications for psychiatric symptoms and decision making.
http://www-psych.stanford.edu/~span
http://www-psych.stanford.edu/~span
G protein coupled receptors (GPCRs) are the largest family of receptors for neurotransmitters in the human genome. We study the structure and mechanism of activation of GPCRs using a variety of biochemical and biophysical approaches including crystallography, NMR and fluorescence spectroscopy.
http://www.med.stanford.edu/kobilkalab/
http://www.med.stanford.edu/kobilkalab/
Cellular mechanisms which monitor protein biogenesis and ensure that only properly folded and assembled proteins are deployed within the cell. Genetic biochemical and cell biological approaches are used to identify the machinery involved in recognizing and destroying misfolded proteins. Molecular mechanisms of neurodegenerative diseases, particular emphasis on Huntington's disease, Alzheimer's disease ALS and prion encephelopathies
http://www.stanford.edu/group/kopito/index.html
http://www.stanford.edu/group/kopito/index.html
In vivo visualization and control of neural circuits for the development of neurological therapeutics.
http://www.ee.ucla.edu/~jhlgroup/
http://www.ee.ucla.edu/~jhlgroup/
Calcium signaling by ion channels and cellular organelles; store-operated channels; calcium control of gene expression.
http://med.stanford.edu/profiles/neuroscience/faculty/Richard_Lewis
http://med.stanford.edu/profiles/neuroscience/faculty/Richard_Lewis
Research in the Vision Lab focus on two intimately connected branches of vision research: computer vision and human vision. In both fields, we are intrigued by visual functionalities that give rise to semantically meaningful interpretations of the visual world. In computer vision, we aspire to build intelligent visual algorithms that perform important visual perception tasks such as object recognition, scene categorization, integrative scene understanding, human motion recognition, etc. In human vision, our curiosity leads us to study the underlying neural mechanisms that enable the human visual system to perform high level visual tasks with amazing speed and efficiency. We use psychophysics experiments, fMRI and computational modeling methods to tackle these extremely interesting yet challenging problems.
http://vision.stanford.edu
http://vision.stanford.edu
My scientific and clinical efforts are inspired by the hope that one day, our studies will lead to effective treatment for optic nerve conditions like anterior ischemic optic neuropathy (AION), the most common acute optic neuropathy in patients over 50 years old, and glaucoma, the most common chronic optic neuropathy in the world. Both diseases result from damage to the axons at the beginning of the optic nerve called the optic nerve head, where 1.2 million axons converge, take a 90-degree turn to form bundles of axons that connect with other visual areas of the brain. Through mechanisms we do not understand, both ischemic optic neuropathy and glaucoma affect predominantly older adults, suggesting an age-dependent susceptibility of the retinal ganglion cell axons at the optic nerve head. AION can be successfully modeled in mouse using laser-stimulated photochemical thrombosis of the small peri-papillary vessels. Using this model, we have evaluated key events following AION and identified potential targets and treatment modalities. We are studying potential neuroprotective agents that may salvage retinal ganglion cells or optic nerve following injury, and we use in vitro as well as non-invasive in vivo imaging techniques including spectral-domain optical coherence tomography and fluorescence confocal scanning laser ophthalmoscopy. We are also studying the potential of using stem cell transplantation to treat AION since the central nervous system has limited repair and regenerative capacity. We have developed substantial experience (> 200 mice) with intravitreal stem cell transplantation using mouse animal models. We show that neural progenitor cells derived from embryonic stem cells (ES-NPCs) can be transplanted into young mouse eyes (P3-5) in large numbers. In contrast, transplantation into adult eyes is much less efficacious, although some cells survived 8-months following transplantation in small numbers. In trying to increase the efficacy of transplantation, we found that retinal laser photocoagulation, a form of controlled injury used routinely to treat some patients with diabetic retinopathy, significantly enhanced the efficacy of transplantation in adult mice. We are continuing our study on manipulations of the neural progenitors as well as the host to optimize regenerative therapy in the adult retina.
http://med.stanford.edu/profiles/Yaping_Liao/
http://med.stanford.edu/profiles/Yaping_Liao/
We have developed fluorecent proteins that mark newly synthesized proteins and are using them to study stimulus-induced local protein translation. We are also developing fluorescent reporters of signaling pathways involved in synaptic growth. We are also developing methods to control protein activity with light or chemicals, and using them to study mechanisms of synaptic plasticity.
http://www.stanford.edu/~mzlin
http://www.stanford.edu/~mzlin
Our studies are focused on elucidation of disease-related signaling mechanisms and development of novel small-molecule strategies for preventing neurodegeneration and promoting neurogenesis and neural function. Disease areas include Alzheimer's and Huntington's.
http://med.stanford.edu/profiles/neuroscience/faculty/Frank_Longo
http://med.stanford.edu/profiles/neuroscience/faculty/Frank_Longo
Neural stem cell behavior; mechanisms of neurodegeneration.
http://med.stanford.edu/profiles/neuroscience/faculty/Bingwei_Lu
http://med.stanford.edu/profiles/neuroscience/faculty/Bingwei_Lu
We use molecular genetics to understand the logic of neural circuit organization and assembly in fruit flies and mice.
http://www.stanford.edu/group/luolab/
http://www.stanford.edu/group/luolab/
Functional and structural neuroimaging of pain from the spinal cord to brain. Central factors contributing to individual differences in pain including cognitive, emotional and decision making. Central plasticity contributing to chronic pain. Real-time fMRI learned control of brain activity and pain.
http://snapl.stanford.edu/
http://snapl.stanford.edu/
Our laboratory uses electrophysiological techniques to study the mechanisms of synaptic transmission and plasticity in the mammalian hippocampus. One of the main focuses in the lab is in the study of synaptic long-term potentiation (LTP).
http://med.stanford.edu/profiles/neuroscience/faculty/Daniel_Madison
http://med.stanford.edu/profiles/neuroscience/faculty/Daniel_Madison
Molecular mechanisms of chloride movement through channels and transporters. Integration of biophysical and electrophysiological methods. Molecular studies of the mechanistic basis of neurostimulation by ultrasound.
http://maduke.stanford.edu/
http://maduke.stanford.edu/
Long-lasting changes in synaptic strength are important for the modification of neural circuits by experience. A major goal of my laboratory is to elucidate the molecular events that trigger various forms of synaptic plasticity and the modifications in synaptic proteins that are responsible for the changes in synaptic efficacy. We also study the behavioral functions of synaptic plasticity in defined circuits using a variety of in vivo molecular manipulations.
http://med.stanford.edu/profiles/neuroscience/faculty/Robert_Malenka
http://med.stanford.edu/profiles/neuroscience/faculty/Robert_Malenka
Two of the main topics of research in my laboratory are dynamics of decision making and learning. I collaborate with the Newsome lab and others to understand how dynamics at the neural level lead to decisions at the level of behavior. We are also interested in the effects of experience on behavior, and how these effects are mediated by changes within the nervous system. We use behavioral experiments and computational models to address these and other issues, and we are open to collaboration with neurophysiologists.
http://psychology.stanford.edu/~jlm
http://psychology.stanford.edu/~jlm
Mechanisms of reward learning and decision-making in humans. Methods include computational modeling and fMRI.
http://www-psych.stanford.edu/~dnl
http://www-psych.stanford.edu/~dnl
We are interested in how individual neurons know where they should sit in the brain and with which neurons they should form specific axonal connections. Our studies explore the molecular and genetic mechanisms by which young neurons locate their correct targets among hundreds of thousands of other neurons in the brain.
http://www.stanford.edu/group/skmlab/
http://www.stanford.edu/group/skmlab/
Theoretical and experimental systems neuroscience - dynamical basis of brain function and dysfunction; functional brain imaging of human cognition and its disruption by mental illness; timing of perceptual and cognitive processes; mathematical models of nonlinear information processing in neural systems.
http://med.stanford.edu/profiles/neuroscience/faculty/Vinod_Menon
http://med.stanford.edu/profiles/neuroscience/faculty/Vinod_Menon
Our laboratory focuses on the molecular mechanisms of postnatal brain development and cancer, with an emphasis neural precursor cells in neuro-oncological diseases. This encompasses the function of normal neural precursor cells after exposure to anti-cancer therapy as well as the role of neural precursor cells in primary neurooncogenesis. I am particulary interested in pediatric brain tumor origins and the molecular signals that drive their growth. As a paradigm of pediatric gliogenesis, I have been focusing on brainstem tumors, whose spatial and temporal specificity bespeak an underlying developmental cause.
https://med.stanford.edu/profiles/stanford/Michelle_Monje-Deisseroth
https://med.stanford.edu/profiles/stanford/Michelle_Monje-Deisseroth
Neural Circuits of Behavior; Mechanisms of visual perception and cognition; visuomotor integration; control of movement.
http://med.stanford.edu/profiles/neuroscience/faculty/Tirin_Moore
http://med.stanford.edu/profiles/neuroscience/faculty/Tirin_Moore
Our long-term goal is dedicated to understanding the genetics of deafness at the molecular level using human and mouse genetics. Our research program focuses on two main areas: neural circuit development related to thyroid hormone, and development and function of the hair cell through action of motor molecules with scaffolding proteins.
Our ongoing research investigates the role of thyroid hormone for the timely coordination of a complex set of neural differentiation events in the maturing cochlea. This includes research on the mechanisms that prompt the progression of axonal projections and synapse formation. We are using mouse genetic manipulations and a variety of molecular and physiological approaches to identify new genes involved in regulating cochlear hair cell innervation.
The second line of research focuses on functional characterization of myosin motors in establishing and maintaining normal hearing and/or vision. Several myosin genes are involved in syndromic and non-syndromic forms of deafness (http://webhost.ua.ac.be/hhh/) but the physiological function of other myosins remains unexplored. The discovery of new molecular motors that are involved in sensory perception and knowledge of their specific roles and cargos will lead to better understanding the differentiation and the function of inner ear hair cells. Genes encoding proteins that interact with myosins comprise another important group of deafness loci. We are using mouse genetic technology to elucidate the role of these motor molecules in hair cell function.
In the long term we are interested in translating the lessons learned in mice to understanding human deafness. We anticipate that the mouse and human genetic studies will be synergistic in advancing our understanding of hearing loss.
Neural processes that mediate visual perception and visually guided behavior.
http://med.stanford.edu/profiles/neuroscience/faculty/William_Newsome
http://med.stanford.edu/profiles/neuroscience/faculty/William_Newsome
Our overarching research focus is on "spatial vision" --- our ability to sense the structure and layout of objects in the world through encoding contrast, pattern, motion and depth. We utilize direct, but non-invasive measures of the brain's electrical activity, Visual Evoked Potentials (VEPs), in addition to functional imaging and behavioral measures to study how the brain processes visual images. As part of our research, we develop new methods for recording and analyzing brain activity, with an emphasis on dynamics. The lab has special interesting in the role of visual experience during development, because experience during development profoundly influences brain structure and function.
Neural precursor cells and the production of new neurons. Local cues that regulate precursor activity. How this information is used to recruit cells for CNS repair or to interrupt precursor signaling once it has gone awry in malignant growth.
http://med.stanford.edu/profiles/neuroscience/faculty/Theo_Palmer
http://med.stanford.edu/profiles/neuroscience/faculty/Theo_Palmer
The core interest that guides our research program is to understand the development of typical and atypical patterns of social behavior. This interest is manifested in two overlapping lines of research: (1) Studies of neuropeptides (e.g., oxytocin) that support social functioning in animals and how alterations in these systems produce social deficits in people with autism; and (2) Investigations of how early social relationships, and their disruption, alter developing neurobiological systems that regulate affect, cognition, and stress reactivity, thereby producing resilient or stress vulnerable organisms.
http://med.stanford.edu/profiles/neuroscience/faculty/Karen_Parker
http://med.stanford.edu/profiles/neuroscience/faculty/Karen_Parker
My lab is involved in electrophysiological recording and electrical stimulation studies in patients implanted with intracranial electrodes for invasive pre surgical monitoring to localize the source of seizures. Our main emphasis is to use electrocorticography and simultaneous EEG/fMRI, and tractography methods to test hypotheses at the level of system neuroscience.
We collect electrophysiological data from the human brain during various cognitive and emotional tasks.
We also study seizure propagation in the human brain, and how the propagation of ictal discharges along specific neuroanatomical circuitries relate to the stereotyped behavior and or thoughts during seizures.
Our motivation is to explore human cognition and how it is broken during seizures.
http://lbcn.stanford.edu/Parvizi_Lab/Welcome.html
http://lbcn.stanford.edu/Parvizi_Lab/Welcome.html
My laboratory is investigating the repair of the injured spinal cord using a number of strategies. These include transplantation of mesenchymal cells, neural stem cells, and glial cellls.In addition we are using gene therapy to improve regeneration of long tract axonal populations by over-expressing or knockdown of important regenerative genes. The laboratory is also investigating Optogenetic tools for improving spinal cord injury and also to investigate the reparative mechanisms of stem cell transplantation.
The Poston Lab seeks to understand the underlying brain circuitry and connectivity associated with movement disorders, such as Parkinson’s disease, and specifically understand the changes in these circuits that lead to specific symptoms, including motor symptoms and cognitive/behavioral symptoms. While current medical and surgical treatments can substantially improve many of the motor symptoms caused by Parkinson’s disease, none of the currently available treatments can cure the disease, alter the disease progression, or significantly treat many of the non-motor symptoms, such as memory problems. Our lab aims to bridge this treatment gap using functional and structural neuroimaging. In addition, we seek to develop novel neuroimaging biomarkers to improve diagnostic accuracy and monitor the efficacy of investigational treatments for Parkinson’s disease and other movement disorders.
molecular regulation of stem cell function; epigenetics of stem cell aging and rejuvenation; stem cell therapies for muscular dystrophies and other muscle disorders; bioengineering of stem cell niches
http://med.stanford.edu/profiles/neuroscience/faculty/Thomas_Rando
http://med.stanford.edu/profiles/neuroscience/faculty/Thomas_Rando
The goal of my research is to determine the role of specific classes of neurons and synapses in shaping the computations performed by the cerebellum. To this end, we are using the latest molecular-genetic approaches for manipulating neural circuits in combination with the detailed behavioral and circuit-level analyses possible in the oculomotor system.
http://med.stanford.edu/profiles/neuroscience/faculty/Jennifer_Raymond
http://med.stanford.edu/profiles/neuroscience/faculty/Jennifer_Raymond
Molecular biology and physiology of neurotransmitter release; neuropathophysiology of lysosomal storage disorders; biosensors.
http://med.stanford.edu/profiles/neuroscience/faculty/Richard_Reimer
http://med.stanford.edu/profiles/neuroscience/faculty/Richard_Reimer
Gene-environment-brain-behavior interactions as elucidated from the study of neurodevelopmental and neuropsychiatric conditions including fragile X syndrome, Williams syndrome, Turner syndrome, velocardiofacial syndrome, autism, preterm birth and other disorders of cognition and behavior. We also study typical and atypical brain development and function with a particular emphasis on measures of resilience. The lab employs comprehensive multi-modal neuroimaging and modeling techniques with identification and measurement of genetic risk factors and neurobehavioral outcome. An interdisciplinary model is emphasized.
http://cibsr.stanford.edu
http://cibsr.stanford.edu
Auditory hair cell mechanotransduction and synaptic transmission.
http://med.stanford.edu/profiles/neuroscience/faculty/Anthony_Ricci
http://med.stanford.edu/profiles/neuroscience/faculty/Anthony_Ricci
How a neuron dies during aging or following various neurological insults; how such neuron death can be accelerated by stress; the design of gene therapy strategies to protect endangered neurons from neurological disease.
http://med.stanford.edu/profiles/neuroscience/faculty/Robert_Sapolsky
http://med.stanford.edu/profiles/neuroscience/faculty/Robert_Sapolsky
Our laboratory investigates the cellular and molecular mechanisms of pain and its control by opioids. When chronic, pain is no longer an essential warning system critical to our survival, but a disease that severely affects the quality of life of many patients. We search to identity the neurons that participate in generating the sensation of pain and to uncover the molecular mechanisms that regulate neural activity in pain circuits. One of our goals is to elucidate the mechanisms by which opioids such as morphine generate analgesia and detrimental side effects, including addiction, to develop more efficient and safer analgesics. To this end we combine a variety of experimental approaches including molecular and cellular biology, neuroanatomy, electrophysiology, optogenetics and behavior.
In vivo fluorescence optical imaging and electrophysiological studies of the mammalian brain towards understanding biophysical aspects of learning and memory. We are developing and applying novel imaging approaches such as multiphoton fluorescence endoscopy for examining individual neurons and dendrites, with emphasis on experiments in awake behaving animals.
http://pyramidal.stanford.edu/
http://pyramidal.stanford.edu/
Genetic regulation of animal development and human disease. 1) We study Hedgehog (Hh) signaling, which controls growth of the cerebellum, and medulloblastoma, the tumors of the cerebellum that occur when Hh signaling is inadequately controlled. 2) Niemann-Pick C (NPC) disease causes Purkinje neurons of the cerebellum to die, and we are studying mechanisms of intracellular transport that underlie normal NPC functions. 3) We have recently discovered that serotonergic signaling in the fly brain is used to control insulin release and thus control of growth, and are studying the circuitry involved as well as identifying new genes required for it. 4) We are using light-activated channel proteins to study the circuitry of Drosophila neuromuscular function and development.
http://med.stanford.edu/labs/matthew_scott/
http://med.stanford.edu/labs/matthew_scott/
The major goal of research is to discover cellular and molecular mechanisms that transform early fetal and neonatal brain circuits into mature patterns of connections during critical periods of development.
http://www.stanford.edu/group/shatzlab/
http://www.stanford.edu/group/shatzlab/
We are interested in understanding how synapses are formed, the final step in wiring a nervous system. In particular, the molecular mechanisms underlying synaptic specify: how neurons recognize each other and how they make decisions about forming synapses between contacting neurites during development. We use molecular, genetic and cell biological tools to study this question in the nematode, C. elegans, which has a very simple nervous system containing only 302 neurons and approximately 6000 synapses.
We are also interested in understanding how synapses are eliminated. During development, synapse formation is always accompanied by synapse elimination. It is the balance between these two events that eventually lead to the maturation of synaptic circuit. Very little is known about synapse elimination. We are using genetic approaches to study this.
Another area of interest is how axons and dendrite polarity is established and maintain.
http://shenlab.stanford.edu
http://shenlab.stanford.edu
Neural prosthetic systems, neural basis
of movement preparation and generation,
population codes and sensorimotor integration.
http://www.stanford.edu/~shenoy/Group.htm
http://www.stanford.edu/~shenoy/Group.htm
The development of novel high-resolution Imaging methods and the exploration of neural circuit connectivity and synapse molecular architectures.
http://smithlab.stanford.edu/
http://smithlab.stanford.edu/
Molecular and cellular mechanisms underlying cerebral ischemia; development of neuroprotective and neurorepair strategies; stem cell transplantation for stroke.
http://med.stanford.edu/profiles/neuroscience/faculty/Gary_Steinberg
http://med.stanford.edu/profiles/neuroscience/faculty/Gary_Steinberg
My laboratory is interested in how presynaptic terminals are formed during synaptogenesis, how presynaptic terminals release neurotransmitters, and how presynaptic terminals degenerate in neurodegenerative disease. To address these questions, we employ diverse approaches ranging from biophysical studies to the physiological and behavioral analyses of mutant mice.
http://www.hhmi.org/research/investigators/sudhof_bio.html
http://www.hhmi.org/research/investigators/sudhof_bio.html
Cognitive neuroscience of memory and cognitive control; prefrontal cortex and medial temporal lobe function; interactions between memory systems.
http://memorylab.stanford.edu/
http://memorylab.stanford.edu/
Modeling visual neurons; development and plasticity of signals in the human visual pathways; current emphases on reading development and cortical plasticity following retinal disease. Magnetic resonance, behavior, and computational methods.
http://white.stanford.edu/wandell.html
http://white.stanford.edu/wandell.html
Mitochondria move and undergo fission and fusion in all eukaryotic cells. The accurate allocation of mitochondria in neurons is particularly critical due to the significance of mitochondria for ATP supply, Ca++ homeostasis and apoptosis and the importance of these functions to the distal extremities of neurons. In addition, defective mitochondria, which can be highly deleterious to a cell because of their output of reactive oxygen species, need to be repaired by fusing with healthy mitochondria or cleared from the cell. Thus mitochondrial cell biology poses critical questions for all cells, but especially for neurons: how the cell sets up an adequate distribution of the organelle; how it sustains mitochondria in the periphery; and how mitochondria are removed after damage. The goal of my research is to understand the regulatory mechanisms controlling mitochondrial dynamics and function and the mechanisms by which even subtle perturbations of these processes may contribute to neurodegenerative disorders.
My lab is generally interested in the mechanisms that determine cell fate identity. Our focus is on epigenetic reprogramming i.e. ways to induce cell fate changes by defined factors such as the reprogramming of somatic cells into pluripotent stem (or iPS) cells. More recently, we have demonstrated that mouse fibroblasts can directly be converted to functional neuronal cells that we termed induced neuronal (iN) cells (Vierbuchen et al., 2010, Nature). The iN cells were generated through expression of the three transcription factors Ascl1, Myt1l, and Brn2. This surprising discovery opened the door to a new area of investigation. We are currently working to apply our finding to human cells, explore the molecular mechanism of the action of the three transcription factors, and determine the neuronal subtype of resulting iN cells. A long term goal is to use this method to evaluate whether iN cells can be used to model neurological diseases.
In addition, the emerging iPS cell technology provides new fascinating translational applications such as patient-specific stem cell therapy or disease phenocopy through differentiation into the neural lineage. Our lab has developed new methods to generate iPS cells from human fibroblasts with defined mutations and explores various technologies to improve gene targeting in human iPS cells with a long term goal to correct disease-causing mutations. This work is made possible through a very generous CIRM grant.
Another interest of the laboratory is to study self-renewal and differentiation in neural stem/progenitor cells and apply these findings to the tumor precursor cells of glioblastoma. This will shed some light into glioma generation and potentially lead to alternative treatment strategies of this devastating brain disease.
http://stemcell.stanford.edu/about/Laboratories/wernig/index.html
http://stemcell.stanford.edu/about/Laboratories/wernig/index.html
Molecular mechanisms of neurodegeneration and Alzheimer’s disease.
http://med.stanford.edu/profiles/neuroscience/faculty/Tony_Wyss-Coray
http://med.stanford.edu/profiles/neuroscience/faculty/Tony_Wyss-Coray
Define the cellular and molecular mechanisms underlying the neurodegeneration associated with cytoskeletal abnormalities
http://med.stanford.edu/profiles/neuroscience/faculty/Yanmin_Yang
http://med.stanford.edu/profiles/neuroscience/faculty/Yanmin_Yang
Pain physiology and molecular biology; herpes vector-directed genetic alteration of sensory neurons; gene therapy for pain; cell transplantation as pain therapy.
http://med.stanford.edu/profiles/neuroscience/faculty/David_Yeomans
http://med.stanford.edu/profiles/neuroscience/faculty/David_Yeomans
My research concerns examination of human and primate circadian rhythms and sleep; notably, the neural mechanisms that underlie wakefulness and circadian photoreception. I am also involved in collaborative efforts in examining the role of sleep disruption in medical pathologies such as Alzheimer's disease, spinal cord injury, and breast cancer.
http://med.stanford.edu/profiles/neuroscience/faculty/Jamie_Zeitzer
http://med.stanford.edu/profiles/neuroscience/faculty/Jamie_Zeitzer
Stroke is one of the leading causes of mortality and morbidity worldwide. Despite extensive research for stroke treatment in the past several decades, few neuroprotectants have been successfully translated from basic research to clinical application. My lab is interested in developing novel therapeutic methods against stroke using various rodent ischemic models, including ischemic postconditioning, remote ischemic pre- or postconditioning, and mild to moderate hypothermia. I hope this research will eventually lead to clinical application. In addition, I am also interested in studying the interaction between brain injury and the immune system (both innate as well as adaptive), including the protective effects of splenectomy.
| Name | Interests | |
| Katrin Andreasson | Mechanisms of neurodegeneration | kandreas@stanford.edu |
| Stephen Baccus | Neural circuits and neural coding; retinal visual processing | baccus@stanford.edu |
| Ben Barres | Neuron-glial interactions in the developing and regenerating CNS | barres@stanford.edu |
| Helen Blau | Cell fate plasticity, stem cell biology, gene therapy, CNS repair | hblau@stanford.edu |
| Kwabena Boahen | Large-scale models of sensory, perceptual and motor systems | boahen@stanford.edu |
| Anne Brunet | Molecular mechanisms of aging in the nervous system | anne.brunet@stanford.edu |
| Axel Brunger | Molecular mechanism of synaptic neurotransmission | brunger@stanford.edu |
| Paul Buckmaster | Hippocampal circuitry, mechanisms of temporal lobe epilepsy | psb@stanford.edu |
| Marion Buckwalter | Neuroinflammation, stroke, functional recovery | marion.buckwalter@stanford.edu |
| Lu Chen | Synaptic signaling and plasticity, synaptic dysfunciton in autism | luchen1@stanford.edu |
| Xiaoke Chen | Brain circuits mediating motivated behaviors and decision-making | xkchen@stanford.edu |
| Yoon-Jae Cho | childhood brain tumors, a5-GABAA receptor biology, RNA helicases in ne | yjcho1@stanford.edu |
| Thomas Clandinin | Genetic approaches to neurodevelopment and behavior | trc@stanford.edu |
| Corrina Darian-Smith | Reorganisation after injury in adult sensorimotor pathways. | cdarian@stanford.edu |
| Luis de Lecea | Neuronal basis of brain arousal | llecea@stanford.edu |
| Karl Deisseroth | Novel optical tools to probe neural circuits in health and disease | deissero@stanford.edu |
| Firdaus Dhabhar | Stress effects on immune function: The good, bad, & beautiful | dhabhar@gmail.com |
| Jun Ding | Neural circuits of movement control in health and movement disorders | dingjun@stanford.edu |
| Ricardo Dolmetsch | Autism, calcium signaling and developmental biology | ricardo.dolmetsch@stanford.edu |
| Amit Etkin | Imaging and manipulating emotion regulation circuits in humans | amitetkin@stanford.edu |
| Russell Fernald | How social behavior influences the brain | rfernald@stanford.edu |
| Surya Ganguli | Theoretical / computational neuroscience. | sganguli@stanford.edu |
| Craig Garner | Molecular mechanisms of synaptic dysfunction in neurodevelopmental dis | cgarner@stanford.edu |
| Joseph Garner | Animal Behavior & Welfare, Repetitive Behavior, Minority students | jgarner@stanford.edu |
| Rona Giffard | astrocytes in cerebral ischemia, heat shock proteins | rgiffard@stanford.edu |
| Lisa Giocomo | Cellular and molecular mechanisms of spatial learning and navigation | giocomo@stanford.edu |
| Aaron Gitler | Mechanisms of human neurodegenerative diseases | agitler@stanford.edu |
| Gary Glover | fMRI acquisition/analysis, experimental design methods | Gary.Glover@stanford.edu |
| Miriam Goodman | Sensory transduction, sensorimotor integration in C. elegans | mbgoodman@stanford.edu |
| Ian Gotlib | Neural, cognitive, and genetic factors in depression and anxiety | ian.gotlib@stanford.edu |
| Kalanit Grill-Spector | Neural correlates of visual perception; | kalanit@stanford.edu |
| James Gross | Emotion regulation in healthy and clinical populations | gross@stanford.edu |
| May Han | Multiple sclerosis | mayhan@stanford.edu |
| H. Craig Heller | Neurobiology of sleep, circadian rhythms, and thermoregulation. | hcheller@stanford.edu |
| Shaul Hestrin | Neocortical Circuits | shaul.hestrin@stanford.edu |
| Ting-Ting Huang | Redox balance in oxidative tissue damage and tissue regeneration | tthuang@stanford.edu |
| John Huguenard | Oscillations/synchronization of neural networks; epilepsy | John.Huguenard@stanford.edu |
| Seung Kim | seungkim@stanford.edu | |
| David Kingsley | Molecular basis of vertebrate evolution | kingsley@cmgm.stanford.edu |
| Eric Knudsen | Circuit and cellular mechanisms of attention | eknudsen@stanford.edu |
| Brian Knutson | Neural basis of emotion, applied to mental health and decision making | knutson@psych.stanford.edu |
| Brian Kobilka | Structural basis for G protein coupled receptor function | kobilka@stanford.edu |
| Ron Kopito | Protein quality control. Molecular genetics and cell biology of neuro | kopito@stanford.edu |
| Jin Hyung Lee | CMP, NBD, SYS | ljinhy@stanford.edu |
| Richard Lewis | Calcium signaling by ion channels and cellular organelles | rslewis@stanford.edu |
| Fei-Fei Li | human vision, high-level visual recognition, computational neuroscienc | feifeili@stanford.edu |
| Y. Joyce Liao | Ischemic optic neuropathy, Stem cell transplantation, Vision electroph | yjliao@stanford.edu |
| Michael Lin | Optogenetics of intracellular signaling pathways | mzlin@stanford.edu |
| Frank Longo | Alzheimer's disease, small molecules growth factor ligands | longo@stanford.edu |
| Bingwei Lu | Neural stem cell biology and neurodegeneration | bingwei@stanford.edu |
| Liqun Luo | Development, organization and function of neuronal circuits | lluo@stanford.edu |
| David Lyons | Behavioral neuroscience | dmlyons@stanford.edu |
| Sean Mackey | Systems neuroscience approaches to human pain research | amorrow@stanford.edu |
| Daniel Madison | Synaptic plasticity and modulation of neuronal excitability. | madison@stanford.edu |
| Merritt Maduke | Molecular mechanisms of ion channels and transporters | maduke@stanford.edu |
| Robert Malenka | synaptic plasticity | malenka@stanford.edu |
| James McClelland | Dynamical models of decision and learning | mcclelland@stanford.edu |
| Samuel McClure | reward learning, decision-making, dopamine function | smcclure@stanford.edu |
| Susan McConnell | Fate determination and migration in mammalian CNS | suemcc@stanford.edu |
| Vinod Menon | Cognitive, developmental & clinical systems neuroscience, Brain Imagin | menon@stanford.edu |
| Michelle Monje-Deisseroth | Brain development, neural stem cells and pediatric brain tumors | mmonje@stanford.edu |
| Tirin Moore | Visuomotor integration, visual perception and cognition | tirin@stanford.edu |
| Mirna Mustapha | Identify genes involved in regulating hair cell innervation | mirnam@stanford.edu |
| William Newsome | Neural basis of visual perception and visually guided cognition | bill@monkeybiz.stanford.edu |
| Anthony Norcia | Vision, development, functional imaging, systems analysis | amnorcia@stanford.edu |
| Theo Palmer | Neural stem cells and inflammation | tpalmer@stanford.edu |
| Karen Parker | Biology of social functioning in monkeys and people with autism | kjparker@stanford.edu |
| Josef Parvizi | Human intracranial cognitive electrophysiological recordings and elect | jparvizi@stanford.edu |
| Giles Plant | Spinal Cord Injury, Visual System Regeneration, Myelination | gplant@stanford.edu |
| Kathleen Poston | Functional Neuroimaging of neurodegenerative disorders | klposton@stanford.edu |
| Thomas Rando | Molecular mechanisms of stem cell fate determination | rando@stanford.edu |
| Jennifer Raymond | Learning & memory; physiology and molecular mechanisms | jenr@stanford.edu |
| Richard Reimer | Neurotransmitter synthesis and packaging | rjreimer@stanford.edu |
| Allan Reiss | Interdisciplinary research, multimodal imaging, brain disorder, typica | reiss@stanford.edu |
| Anthony Ricci | Auditory hair cell mechanotransduction and synaptic transmissio | tricci@ohns.stanford.edu |
| Robert Sapolsky | Neuron death, stress, gene therapy | sapolsky@stanford.edu |
| Gregory Scherrer | Cellular and molecular mechanisms of pain and its control by opioids | gs25@stanford.edu |
| Mark Schnitzer | In vivo two-photon imaging; cerebellar and hippocampal circuits | mschnitz@stanford.edu |
| Matthew Scott | Cerebellum development and cancer, neural control of growth | mscott@stanford.edu |
| Carla Shatz | molecular mechanisms of experience-dependent circuit formation | cshatz@stanford.edu |
| Kang Shen | moleular mechanisms of circuit assembly at the level of synapse | kangshen@stanford.edu |
| Krishna Shenoy | Computational motor neurophysiology, neural prostheses | shenoy@stanford.edu |
| Stephen Smith | Neural Circuit Molecular Architectures | sjsmith@stanford.edu |
| Gary Steinberg | Pathophysiology and treatment of focal and global cerebral ischemia | steinberg@stanford.edu |
| Thomas Sudhof | Formation, function, and dysfunction of synapses | tcs1@stanford.edu |
| Patrick Suppes | Mulitvariate analysis of neural signals | psuppes@stanford.edu |
| Anthony Wagner | Neural basis of memory and executive function | awagner@stanford.edu |
| Brian Wandell | Visual perception, reading development, fMRI, DTI | wandell@stanford.edu |
| Xinnan Wang | Mitochondria in Neuronal Health and Disease | xinnanw@stanford.edu |
| Marius Wernig | Reprogramming, induced neuronal (iN) cells, pluripotent stem cells, di | wernig@stanford.edu |
| Tony Wyss-Coray | Molecular basis of neurodegeneration and Alzheimer's disease | twc@stanford.edu |
| Yanmin Yang | Cytoskeletal function and dysfunction in the nervous system | yyanmin@stanford.edu |
| David Yeomans | Pain: differential activation; gene therapy; sodium channels | dcyeomans@stanford.edu |
| Jamie Zeitzer | Neurobio of sleep and circadian photoreception | jzeitzer@stanford.edu |
| Heng Zhao | stroke, postconditioning, peripheral immune system and brain injury | hzhao@stanford.edu |
Developmental
| Name | Interests | |
|---|---|---|
| Ben Barres | Neuron-glial interactions in the developing and regenerating CNS | barres@stanford.edu |
| Helen Blau | Cell fate plasticity, stem cell biology, gene therapy, CNS repair | hblau@stanford.edu |
| Kwabena Boahen | Large-scale models of sensory, perceptual and motor systems | boahen@stanford.edu |
| Anne Brunet | Molecular mechanisms of aging in the nervous system | anne.brunet@stanford.edu |
| Lu Chen | Synaptic signaling and plasticity, synaptic dysfunciton in autism | luchen1@stanford.edu |
| Yoon-Jae Cho | childhood brain tumors, a5-GABAA receptor biology, RNA helicases in ne | yjcho1@stanford.edu |
| Thomas Clandinin | Genetic approaches to neurodevelopment and behavior | trc@stanford.edu |
| Ricardo Dolmetsch | Autism, calcium signaling and developmental biology | ricardo.dolmetsch@stanford.edu |
| Russell Fernald | How social behavior influences the brain | rfernald@stanford.edu |
| Craig Garner | Molecular mechanisms of synaptic dysfunction in neurodevelopmental dis | cgarner@stanford.edu |
| Joseph Garner | Animal Behavior & Welfare, Repetitive Behavior, Minority students | jgarner@stanford.edu |
| Rona Giffard | astrocytes in cerebral ischemia, heat shock proteins | rgiffard@stanford.edu |
| Gary Glover | fMRI acquisition/analysis, experimental design methods | Gary.Glover@stanford.edu |
| Ian Gotlib | Neural, cognitive, and genetic factors in depression and anxiety | ian.gotlib@stanford.edu |
| H. Craig Heller | Neurobiology of sleep, circadian rhythms, and thermoregulation. | hcheller@stanford.edu |
| David Kingsley | Molecular basis of vertebrate evolution | kingsley@cmgm.stanford.edu |
| Eric Knudsen | Circuit and cellular mechanisms of attention | eknudsen@stanford.edu |
| Bingwei Lu | Neural stem cell biology and neurodegeneration | bingwei@stanford.edu |
| Liqun Luo | Development, organization and function of neuronal circuits | lluo@stanford.edu |
| Daniel Madison | Synaptic plasticity and modulation of neuronal excitability. | madison@stanford.edu |
| Susan McConnell | Fate determination and migration in mammalian CNS | suemcc@stanford.edu |
| Vinod Menon | Cognitive, developmental & clinical systems neuroscience, Brain Imagin | menon@stanford.edu |
| Michelle Monje-Deisseroth | Brain development, neural stem cells and pediatric brain tumors | mmonje@stanford.edu |
| Anthony Norcia | Vision, development, functional imaging, systems analysis | amnorcia@stanford.edu |
| Karen Parker | Biology of social functioning in monkeys and people with autism | kjparker@stanford.edu |
| Thomas Rando | Molecular mechanisms of stem cell fate determination | rando@stanford.edu |
| Allan Reiss | Interdisciplinary research, multimodal imaging, brain disorder, typica | reiss@stanford.edu |
| Anthony Ricci | Auditory hair cell mechanotransduction and synaptic transmissio | tricci@ohns.stanford.edu |
| Matthew Scott | Cerebellum development and cancer, neural control of growth | mscott@stanford.edu |
| Carla Shatz | molecular mechanisms of experience-dependent circuit formation | cshatz@stanford.edu |
| Kang Shen | moleular mechanisms of circuit assembly at the level of synapse | kangshen@stanford.edu |
| Stephen Smith | Neural Circuit Molecular Architectures | sjsmith@stanford.edu |
| Thomas Sudhof | Formation, function, and dysfunction of synapses | tcs1@stanford.edu |
| Brian Wandell | Visual perception, reading development, fMRI, DTI | wandell@stanford.edu |
| Marius Wernig | Reprogramming, induced neuronal (iN) cells, pluripotent stem cells, di | wernig@stanford.edu |
| Yanmin Yang | Cytoskeletal function and dysfunction in the nervous system | yyanmin@stanford.edu |
Computational
| Name | Interests | |
|---|---|---|
| Stephen Baccus | Neural circuits and neural coding; retinal visual processing | baccus@stanford.edu |
| Kwabena Boahen | Large-scale models of sensory, perceptual and motor systems | boahen@stanford.edu |
| Axel Brunger | Molecular mechanism of synaptic neurotransmission | brunger@stanford.edu |
| Yoon-Jae Cho | childhood brain tumors, a5-GABAA receptor biology, RNA helicases in ne | yjcho1@stanford.edu |
| Karl Deisseroth | Novel optical tools to probe neural circuits in health and disease | deissero@stanford.edu |
| Ricardo Dolmetsch | Autism, calcium signaling and developmental biology | ricardo.dolmetsch@stanford.edu |
| Amit Etkin | Imaging and manipulating emotion regulation circuits in humans | amitetkin@stanford.edu |
| Surya Ganguli | Theoretical / computational neuroscience. | sganguli@stanford.edu |
| Rona Giffard | astrocytes in cerebral ischemia, heat shock proteins | rgiffard@stanford.edu |
| Gary Glover | fMRI acquisition/analysis, experimental design methods | Gary.Glover@stanford.edu |
| Kalanit Grill-Spector | Neural correlates of visual perception; | kalanit@stanford.edu |
| John Huguenard | Oscillations/synchronization of neural networks; epilepsy | John.Huguenard@stanford.edu |
| David Kingsley | Molecular basis of vertebrate evolution | kingsley@cmgm.stanford.edu |
| Fei-Fei Li | human vision, high-level visual recognition, computational neuroscienc | feifeili@stanford.edu |
| Sean Mackey | Systems neuroscience approaches to human pain research | amorrow@stanford.edu |
| James McClelland | Dynamical models of decision and learning | mcclelland@stanford.edu |
| Samuel McClure | reward learning, decision-making, dopamine function | smcclure@stanford.edu |
| Vinod Menon | Cognitive, developmental & clinical systems neuroscience, Brain Imagin | menon@stanford.edu |
| Tirin Moore | Visuomotor integration, visual perception and cognition | tirin@stanford.edu |
| Anthony Norcia | Vision, development, functional imaging, systems analysis | amnorcia@stanford.edu |
| Josef Parvizi | Human intracranial cognitive electrophysiological recordings and elect | jparvizi@stanford.edu |
| Jennifer Raymond | Learning & memory; physiology and molecular mechanisms | jenr@stanford.edu |
| Allan Reiss | Interdisciplinary research, multimodal imaging, brain disorder, typica | reiss@stanford.edu |
| Anthony Ricci | Auditory hair cell mechanotransduction and synaptic transmissio | tricci@ohns.stanford.edu |
| Mark Schnitzer | In vivo two-photon imaging; cerebellar and hippocampal circuits | mschnitz@stanford.edu |
| Krishna Shenoy | Computational motor neurophysiology, neural prostheses | shenoy@stanford.edu |
| Stephen Smith | Neural Circuit Molecular Architectures | sjsmith@stanford.edu |
| Patrick Suppes | Mulitvariate analysis of neural signals | psuppes@stanford.edu |
| Brian Wandell | Visual perception, reading development, fMRI, DTI | wandell@stanford.edu |
Excitability
| Name | Interests | |
|---|---|---|
| Ben Barres | Neuron-glial interactions in the developing and regenerating CNS | barres@stanford.edu |
| Paul Buckmaster | Hippocampal circuitry, mechanisms of temporal lobe epilepsy | psb@stanford.edu |
| Lu Chen | Synaptic signaling and plasticity, synaptic dysfunciton in autism | luchen1@stanford.edu |
| Karl Deisseroth | Novel optical tools to probe neural circuits in health and disease | deissero@stanford.edu |
| Jun Ding | Neural circuits of movement control in health and movement disorders | dingjun@stanford.edu |
| Ricardo Dolmetsch | Autism, calcium signaling and developmental biology | ricardo.dolmetsch@stanford.edu |
| Miriam Goodman | Sensory transduction, sensorimotor integration in C. elegans | mbgoodman@stanford.edu |
| Shaul Hestrin | Neocortical Circuits | shaul.hestrin@stanford.edu |
| John Huguenard | Oscillations/synchronization of neural networks; epilepsy | John.Huguenard@stanford.edu |
| Richard Lewis | Calcium signaling by ion channels and cellular organelles | rslewis@stanford.edu |
| Sean Mackey | Systems neuroscience approaches to human pain research | amorrow@stanford.edu |
| Daniel Madison | Synaptic plasticity and modulation of neuronal excitability. | madison@stanford.edu |
| Merritt Maduke | Molecular mechanisms of ion channels and transporters | maduke@stanford.edu |
| Robert Malenka | synaptic plasticity | malenka@stanford.edu |
| Anthony Ricci | Auditory hair cell mechanotransduction and synaptic transmissio | tricci@ohns.stanford.edu |
| Gregory Scherrer | Cellular and molecular mechanisms of pain and its control by opioids | gs25@stanford.edu |
| Matthew Scott | Cerebellum development and cancer, neural control of growth | mscott@stanford.edu |
| Carla Shatz | molecular mechanisms of experience-dependent circuit formation | cshatz@stanford.edu |
| Stephen Smith | Neural Circuit Molecular Architectures | sjsmith@stanford.edu |
| Thomas Sudhof | Formation, function, and dysfunction of synapses | tcs1@stanford.edu |
| David Yeomans | Pain: differential activation; gene therapy; sodium channels | dcyeomans@stanford.edu |
Disease
| Name | Interests | |
|---|---|---|
| Katrin Andreasson | Mechanisms of neurodegeneration | kandreas@stanford.edu |
| Ben Barres | Neuron-glial interactions in the developing and regenerating CNS | barres@stanford.edu |
| Helen Blau | Cell fate plasticity, stem cell biology, gene therapy, CNS repair | hblau@stanford.edu |
| Anne Brunet | Molecular mechanisms of aging in the nervous system | anne.brunet@stanford.edu |
| Axel Brunger | Molecular mechanism of synaptic neurotransmission | brunger@stanford.edu |
| Paul Buckmaster | Hippocampal circuitry, mechanisms of temporal lobe epilepsy | psb@stanford.edu |
| Marion Buckwalter | Neuroinflammation, stroke, functional recovery | marion.buckwalter@stanford.edu |
| Lu Chen | Synaptic signaling and plasticity, synaptic dysfunciton in autism | luchen1@stanford.edu |
| Yoon-Jae Cho | childhood brain tumors, a5-GABAA receptor biology, RNA helicases in ne | yjcho1@stanford.edu |
| Corrina Darian-Smith | Reorganisation after injury in adult sensorimotor pathways. | cdarian@stanford.edu |
| Luis de Lecea | Neuronal basis of brain arousal | llecea@stanford.edu |
| Karl Deisseroth | Novel optical tools to probe neural circuits in health and disease | deissero@stanford.edu |
| Firdaus Dhabhar | Stress effects on immune function: The good, bad, & beautiful | dhabhar@gmail.com |
| Jun Ding | Neural circuits of movement control in health and movement disorders | dingjun@stanford.edu |
| Ricardo Dolmetsch | Autism, calcium signaling and developmental biology | ricardo.dolmetsch@stanford.edu |
| Amit Etkin | Imaging and manipulating emotion regulation circuits in humans | amitetkin@stanford.edu |
| Joseph Garner | Animal Behavior & Welfare, Repetitive Behavior, Minority students | jgarner@stanford.edu |
| Rona Giffard | astrocytes in cerebral ischemia, heat shock proteins | rgiffard@stanford.edu |
| Aaron Gitler | Mechanisms of human neurodegenerative diseases | agitler@stanford.edu |
| Gary Glover | fMRI acquisition/analysis, experimental design methods | Gary.Glover@stanford.edu |
| Ian Gotlib | Neural, cognitive, and genetic factors in depression and anxiety | ian.gotlib@stanford.edu |
| May Han | Multiple sclerosis | mayhan@stanford.edu |
| H. Craig Heller | Neurobiology of sleep, circadian rhythms, and thermoregulation. | hcheller@stanford.edu |
| Ting-Ting Huang | Redox balance in oxidative tissue damage and tissue regeneration | tthuang@stanford.edu |
| John Huguenard | Oscillations/synchronization of neural networks; epilepsy | John.Huguenard@stanford.edu |
| Brian Knutson | Neural basis of emotion, applied to mental health and decision making | knutson@psych.stanford.edu |
| Ron Kopito | Protein quality control. Molecular genetics and cell biology of neuro | kopito@stanford.edu |
| Y. Joyce Liao | Ischemic optic neuropathy, Stem cell transplantation, Vision electroph | yjliao@stanford.edu |
| Frank Longo | Alzheimer's disease, small molecules growth factor ligands | longo@stanford.edu |
| Bingwei Lu | Neural stem cell biology and neurodegeneration | bingwei@stanford.edu |
| Sean Mackey | Systems neuroscience approaches to human pain research | amorrow@stanford.edu |
| Daniel Madison | Synaptic plasticity and modulation of neuronal excitability. | madison@stanford.edu |
| Merritt Maduke | Molecular mechanisms of ion channels and transporters | maduke@stanford.edu |
| Robert Malenka | synaptic plasticity | malenka@stanford.edu |
| Vinod Menon | Cognitive, developmental & clinical systems neuroscience, Brain Imagin | menon@stanford.edu |
| Michelle Monje-Deisseroth | Brain development, neural stem cells and pediatric brain tumors | mmonje@stanford.edu |
| Tirin Moore | Visuomotor integration, visual perception and cognition | tirin@stanford.edu |
| Mirna Mustapha | Identify genes involved in regulating hair cell innervation | mirnam@stanford.edu |
| Anthony Norcia | Vision, development, functional imaging, systems analysis | amnorcia@stanford.edu |
| Karen Parker | Biology of social functioning in monkeys and people with autism | kjparker@stanford.edu |
| Josef Parvizi | Human intracranial cognitive electrophysiological recordings and elect | jparvizi@stanford.edu |
| Giles Plant | Spinal Cord Injury, Visual System Regeneration, Myelination | gplant@stanford.edu |
| Kathleen Poston | Functional Neuroimaging of neurodegenerative disorders | klposton@stanford.edu |
| Thomas Rando | Molecular mechanisms of stem cell fate determination | rando@stanford.edu |
| Richard Reimer | Neurotransmitter synthesis and packaging | rjreimer@stanford.edu |
| Allan Reiss | Interdisciplinary research, multimodal imaging, brain disorder, typica | reiss@stanford.edu |
| Robert Sapolsky | Neuron death, stress, gene therapy | sapolsky@stanford.edu |
| Gregory Scherrer | Cellular and molecular mechanisms of pain and its control by opioids | gs25@stanford.edu |
| Mark Schnitzer | In vivo two-photon imaging; cerebellar and hippocampal circuits | mschnitz@stanford.edu |
| Matthew Scott | Cerebellum development and cancer, neural control of growth | mscott@stanford.edu |
| Stephen Smith | Neural Circuit Molecular Architectures | sjsmith@stanford.edu |
| Thomas Sudhof | Formation, function, and dysfunction of synapses | tcs1@stanford.edu |
| Anthony Wagner | Neural basis of memory and executive function | awagner@stanford.edu |
| Brian Wandell | Visual perception, reading development, fMRI, DTI | wandell@stanford.edu |
| Xinnan Wang | Mitochondria in Neuronal Health and Disease | xinnanw@stanford.edu |
| Marius Wernig | Reprogramming, induced neuronal (iN) cells, pluripotent stem cells, di | wernig@stanford.edu |
| Tony Wyss-Coray | Molecular basis of neurodegeneration and Alzheimer's disease | twc@stanford.edu |
| Yanmin Yang | Cytoskeletal function and dysfunction in the nervous system | yyanmin@stanford.edu |
| David Yeomans | Pain: differential activation; gene therapy; sodium channels | dcyeomans@stanford.edu |
| Jamie Zeitzer | Neurobio of sleep and circadian photoreception | jzeitzer@stanford.edu |
| Heng Zhao | stroke, postconditioning, peripheral immune system and brain injury | hzhao@stanford.edu |
Systems
| Name | Interests | |
|---|---|---|
| Stephen Baccus | Neural circuits and neural coding; retinal visual processing | baccus@stanford.edu |
| Kwabena Boahen | Large-scale models of sensory, perceptual and motor systems | boahen@stanford.edu |
| Paul Buckmaster | Hippocampal circuitry, mechanisms of temporal lobe epilepsy | psb@stanford.edu |
| Xiaoke Chen | Brain circuits mediating motivated behaviors and decision-making | xkchen@stanford.edu |
| Thomas Clandinin | Genetic approaches to neurodevelopment and behavior | trc@stanford.edu |
| Corrina Darian-Smith | Reorganisation after injury in adult sensorimotor pathways. | cdarian@stanford.edu |
| Luis de Lecea | Neuronal basis of brain arousal | llecea@stanford.edu |
| Karl Deisseroth | Novel optical tools to probe neural circuits in health and disease | deissero@stanford.edu |
| Firdaus Dhabhar | Stress effects on immune function: The good, bad, & beautiful | dhabhar@gmail.com |
| Amit Etkin | Imaging and manipulating emotion regulation circuits in humans | amitetkin@stanford.edu |
| Russell Fernald | How social behavior influences the brain | rfernald@stanford.edu |
| Surya Ganguli | Theoretical / computational neuroscience. | sganguli@stanford.edu |
| Joseph Garner | Animal Behavior & Welfare, Repetitive Behavior, Minority students | jgarner@stanford.edu |
| Lisa Giocomo | Cellular and molecular mechanisms of spatial learning and navigation | giocomo@stanford.edu |
| Gary Glover | fMRI acquisition/analysis, experimental design methods | Gary.Glover@stanford.edu |
| Ian Gotlib | Neural, cognitive, and genetic factors in depression and anxiety | ian.gotlib@stanford.edu |
| Kalanit Grill-Spector | Neural correlates of visual perception; | kalanit@stanford.edu |
| James Gross | Emotion regulation in healthy and clinical populations | gross@stanford.edu |
| H. Craig Heller | Neurobiology of sleep, circadian rhythms, and thermoregulation. | hcheller@stanford.edu |
| Shaul Hestrin | Neocortical Circuits | shaul.hestrin@stanford.edu |
| Eric Knudsen | Circuit and cellular mechanisms of attention | eknudsen@stanford.edu |
| Brian Knutson | Neural basis of emotion, applied to mental health and decision making | knutson@psych.stanford.edu |
| Fei-Fei Li | human vision, high-level visual recognition, computational neuroscienc | feifeili@stanford.edu |
| Y. Joyce Liao | Ischemic optic neuropathy, Stem cell transplantation, Vision electroph | yjliao@stanford.edu |
| Liqun Luo | Development, organization and function of neuronal circuits | lluo@stanford.edu |
| Sean Mackey | Systems neuroscience approaches to human pain research | amorrow@stanford.edu |
| Daniel Madison | Synaptic plasticity and modulation of neuronal excitability. | madison@stanford.edu |
| Robert Malenka | synaptic plasticity | malenka@stanford.edu |
| James McClelland | Dynamical models of decision and learning | mcclelland@stanford.edu |
| Samuel McClure | reward learning, decision-making, dopamine function | smcclure@stanford.edu |
| Vinod Menon | Cognitive, developmental & clinical systems neuroscience, Brain Imagin | menon@stanford.edu |
| Tirin Moore | Visuomotor integration, visual perception and cognition | tirin@stanford.edu |
| William Newsome | Neural basis of visual perception and visually guided cognition | bill@monkeybiz.stanford.edu |
| Anthony Norcia | Vision, development, functional imaging, systems analysis | amnorcia@stanford.edu |
| Karen Parker | Biology of social functioning in monkeys and people with autism | kjparker@stanford.edu |
| Josef Parvizi | Human intracranial cognitive electrophysiological recordings and elect | jparvizi@stanford.edu |
| Kathleen Poston | Functional Neuroimaging of neurodegenerative disorders | klposton@stanford.edu |
| Jennifer Raymond | Learning & memory; physiology and molecular mechanisms | jenr@stanford.edu |
| Allan Reiss | Interdisciplinary research, multimodal imaging, brain disorder, typica | reiss@stanford.edu |
| Robert Sapolsky | Neuron death, stress, gene therapy | sapolsky@stanford.edu |
| Gregory Scherrer | Cellular and molecular mechanisms of pain and its control by opioids | gs25@stanford.edu |
| Mark Schnitzer | In vivo two-photon imaging; cerebellar and hippocampal circuits | mschnitz@stanford.edu |
| Matthew Scott | Cerebellum development and cancer, neural control of growth | mscott@stanford.edu |
| Carla Shatz | molecular mechanisms of experience-dependent circuit formation | cshatz@stanford.edu |
| Krishna Shenoy | Computational motor neurophysiology, neural prostheses | shenoy@stanford.edu |
| Stephen Smith | Neural Circuit Molecular Architectures | sjsmith@stanford.edu |
| Gary Steinberg | Pathophysiology and treatment of focal and global cerebral ischemia | steinberg@stanford.edu |
| Thomas Sudhof | Formation, function, and dysfunction of synapses | tcs1@stanford.edu |
| Patrick Suppes | Mulitvariate analysis of neural signals | psuppes@stanford.edu |
| Anthony Wagner | Neural basis of memory and executive function | awagner@stanford.edu |
| Brian Wandell | Visual perception, reading development, fMRI, DTI | wandell@stanford.edu |
| David Yeomans | Pain: differential activation; gene therapy; sodium channels | dcyeomans@stanford.edu |
| Jamie Zeitzer | Neurobio of sleep and circadian photoreception | jzeitzer@stanford.edu |
Cellular
| Name | Interests | |
|---|---|---|
| Katrin Andreasson | Mechanisms of neurodegeneration | kandreas@stanford.edu |
| Stephen Baccus | Neural circuits and neural coding; retinal visual processing | baccus@stanford.edu |
| Ben Barres | Neuron-glial interactions in the developing and regenerating CNS | barres@stanford.edu |
| Helen Blau | Cell fate plasticity, stem cell biology, gene therapy, CNS repair | hblau@stanford.edu |
| Anne Brunet | Molecular mechanisms of aging in the nervous system | anne.brunet@stanford.edu |
| Axel Brunger | Molecular mechanism of synaptic neurotransmission | brunger@stanford.edu |
| Paul Buckmaster | Hippocampal circuitry, mechanisms of temporal lobe epilepsy | psb@stanford.edu |
| Marion Buckwalter | Neuroinflammation, stroke, functional recovery | marion.buckwalter@stanford.edu |
| Lu Chen | Synaptic signaling and plasticity, synaptic dysfunciton in autism | luchen1@stanford.edu |
| Xiaoke Chen | Brain circuits mediating motivated behaviors and decision-making | xkchen@stanford.edu |
| Yoon-Jae Cho | childhood brain tumors, a5-GABAA receptor biology, RNA helicases in ne | yjcho1@stanford.edu |
| Thomas Clandinin | Genetic approaches to neurodevelopment and behavior | trc@stanford.edu |
| Corrina Darian-Smith | Reorganisation after injury in adult sensorimotor pathways. | cdarian@stanford.edu |
| Karl Deisseroth | Novel optical tools to probe neural circuits in health and disease | deissero@stanford.edu |
| Firdaus Dhabhar | Stress effects on immune function: The good, bad, & beautiful | dhabhar@gmail.com |
| Jun Ding | Neural circuits of movement control in health and movement disorders | dingjun@stanford.edu |
| Ricardo Dolmetsch | Autism, calcium signaling and developmental biology | ricardo.dolmetsch@stanford.edu |
| Russell Fernald | How social behavior influences the brain | rfernald@stanford.edu |
| Craig Garner | Molecular mechanisms of synaptic dysfunction in neurodevelopmental dis | cgarner@stanford.edu |
| Rona Giffard | astrocytes in cerebral ischemia, heat shock proteins | rgiffard@stanford.edu |
| Lisa Giocomo | Cellular and molecular mechanisms of spatial learning and navigation | giocomo@stanford.edu |
| Aaron Gitler | Mechanisms of human neurodegenerative diseases | agitler@stanford.edu |
| Miriam Goodman | Sensory transduction, sensorimotor integration in C. elegans | mbgoodman@stanford.edu |
| H. Craig Heller | Neurobiology of sleep, circadian rhythms, and thermoregulation. | hcheller@stanford.edu |
| Shaul Hestrin | Neocortical Circuits | shaul.hestrin@stanford.edu |
| Ting-Ting Huang | Redox balance in oxidative tissue damage and tissue regeneration | tthuang@stanford.edu |
| John Huguenard | Oscillations/synchronization of neural networks; epilepsy | John.Huguenard@stanford.edu |
| Eric Knudsen | Circuit and cellular mechanisms of attention | eknudsen@stanford.edu |
| Brian Kobilka | Structural basis for G protein coupled receptor function | kobilka@stanford.edu |
| Ron Kopito | Protein quality control. Molecular genetics and cell biology of neuro | kopito@stanford.edu |
| Richard Lewis | Calcium signaling by ion channels and cellular organelles | rslewis@stanford.edu |
| Y. Joyce Liao | Ischemic optic neuropathy, Stem cell transplantation, Vision electroph | yjliao@stanford.edu |
| Michael Lin | Optogenetics of intracellular signaling pathways | mzlin@stanford.edu |
| Bingwei Lu | Neural stem cell biology and neurodegeneration | bingwei@stanford.edu |
| Liqun Luo | Development, organization and function of neuronal circuits | lluo@stanford.edu |
| Daniel Madison | Synaptic plasticity and modulation of neuronal excitability. | madison@stanford.edu |
| Robert Malenka | synaptic plasticity | malenka@stanford.edu |
| Susan McConnell | Fate determination and migration in mammalian CNS | suemcc@stanford.edu |
| Michelle Monje-Deisseroth | Brain development, neural stem cells and pediatric brain tumors | mmonje@stanford.edu |
| Tirin Moore | Visuomotor integration, visual perception and cognition | tirin@stanford.edu |
| Theo Palmer | Neural stem cells and inflammation | tpalmer@stanford.edu |
| Giles Plant | Spinal Cord Injury, Visual System Regeneration, Myelination | gplant@stanford.edu |
| Thomas Rando | Molecular mechanisms of stem cell fate determination | rando@stanford.edu |
| Richard Reimer | Neurotransmitter synthesis and packaging | rjreimer@stanford.edu |
| Anthony Ricci | Auditory hair cell mechanotransduction and synaptic transmissio | tricci@ohns.stanford.edu |
| Robert Sapolsky | Neuron death, stress, gene therapy | sapolsky@stanford.edu |
| Gregory Scherrer | Cellular and molecular mechanisms of pain and its control by opioids | gs25@stanford.edu |
| Mark Schnitzer | In vivo two-photon imaging; cerebellar and hippocampal circuits | mschnitz@stanford.edu |
| Matthew Scott | Cerebellum development and cancer, neural control of growth | mscott@stanford.edu |
| Carla Shatz | molecular mechanisms of experience-dependent circuit formation | cshatz@stanford.edu |
| Kang Shen | moleular mechanisms of circuit assembly at the level of synapse | kangshen@stanford.edu |
| Stephen Smith | Neural Circuit Molecular Architectures | sjsmith@stanford.edu |
| Gary Steinberg | Pathophysiology and treatment of focal and global cerebral ischemia | steinberg@stanford.edu |
| Thomas Sudhof | Formation, function, and dysfunction of synapses | tcs1@stanford.edu |
| Xinnan Wang | Mitochondria in Neuronal Health and Disease | xinnanw@stanford.edu |
| Marius Wernig | Reprogramming, induced neuronal (iN) cells, pluripotent stem cells, di | wernig@stanford.edu |
| Tony Wyss-Coray | Molecular basis of neurodegeneration and Alzheimer's disease | twc@stanford.edu |
| Yanmin Yang | Cytoskeletal function and dysfunction in the nervous system | yyanmin@stanford.edu |
| Heng Zhao | stroke, postconditioning, peripheral immune system and brain injury | hzhao@stanford.edu |
Molecular
| Name | Interests | |
|---|---|---|
| Katrin Andreasson | Mechanisms of neurodegeneration | kandreas@stanford.edu |
| Ben Barres | Neuron-glial interactions in the developing and regenerating CNS | barres@stanford.edu |
| Helen Blau | Cell fate plasticity, stem cell biology, gene therapy, CNS repair | hblau@stanford.edu |
| Anne Brunet | Molecular mechanisms of aging in the nervous system | anne.brunet@stanford.edu |
| Axel Brunger | Molecular mechanism of synaptic neurotransmission | brunger@stanford.edu |
| Marion Buckwalter | Neuroinflammation, stroke, functional recovery | marion.buckwalter@stanford.edu |
| Lu Chen | Synaptic signaling and plasticity, synaptic dysfunciton in autism | luchen1@stanford.edu |
| Xiaoke Chen | Brain circuits mediating motivated behaviors and decision-making | xkchen@stanford.edu |
| Yoon-Jae Cho | childhood brain tumors, a5-GABAA receptor biology, RNA helicases in ne | yjcho1@stanford.edu |
| Thomas Clandinin | Genetic approaches to neurodevelopment and behavior | trc@stanford.edu |
| Luis de Lecea | Neuronal basis of brain arousal | llecea@stanford.edu |
| Karl Deisseroth | Novel optical tools to probe neural circuits in health and disease | deissero@stanford.edu |
| Firdaus Dhabhar | Stress effects on immune function: The good, bad, & beautiful | dhabhar@gmail.com |
| Jun Ding | Neural circuits of movement control in health and movement disorders | dingjun@stanford.edu |
| Ricardo Dolmetsch | Autism, calcium signaling and developmental biology | ricardo.dolmetsch@stanford.edu |
| Russell Fernald | How social behavior influences the brain | rfernald@stanford.edu |
| Craig Garner | Molecular mechanisms of synaptic dysfunction in neurodevelopmental dis | cgarner@stanford.edu |
| Rona Giffard | astrocytes in cerebral ischemia, heat shock proteins | rgiffard@stanford.edu |
| Aaron Gitler | Mechanisms of human neurodegenerative diseases | agitler@stanford.edu |
| Miriam Goodman | Sensory transduction, sensorimotor integration in C. elegans | mbgoodman@stanford.edu |
| Shaul Hestrin | Neocortical Circuits | shaul.hestrin@stanford.edu |
| Ting-Ting Huang | Redox balance in oxidative tissue damage and tissue regeneration | tthuang@stanford.edu |
| John Huguenard | Oscillations/synchronization of neural networks; epilepsy | John.Huguenard@stanford.edu |
| David Kingsley | Molecular basis of vertebrate evolution | kingsley@cmgm.stanford.edu |
| Brian Kobilka | Structural basis for G protein coupled receptor function | kobilka@stanford.edu |
| Ron Kopito | Protein quality control. Molecular genetics and cell biology of neuro | kopito@stanford.edu |
| Richard Lewis | Calcium signaling by ion channels and cellular organelles | rslewis@stanford.edu |
| Y. Joyce Liao | Ischemic optic neuropathy, Stem cell transplantation, Vision electroph | yjliao@stanford.edu |
| Michael Lin | Optogenetics of intracellular signaling pathways | mzlin@stanford.edu |
| Frank Longo | Alzheimer's disease, small molecules growth factor ligands | longo@stanford.edu |
| Bingwei Lu | Neural stem cell biology and neurodegeneration | bingwei@stanford.edu |
| Liqun Luo | Development, organization and function of neuronal circuits | lluo@stanford.edu |
| Merritt Maduke | Molecular mechanisms of ion channels and transporters | maduke@stanford.edu |
| Robert Malenka | synaptic plasticity | malenka@stanford.edu |
| Susan McConnell | Fate determination and migration in mammalian CNS | suemcc@stanford.edu |
| Michelle Monje-Deisseroth | Brain development, neural stem cells and pediatric brain tumors | mmonje@stanford.edu |
| Mirna Mustapha | Identify genes involved in regulating hair cell innervation | mirnam@stanford.edu |
| Giles Plant | Spinal Cord Injury, Visual System Regeneration, Myelination | gplant@stanford.edu |
| Thomas Rando | Molecular mechanisms of stem cell fate determination | rando@stanford.edu |
| Jennifer Raymond | Learning & memory; physiology and molecular mechanisms | jenr@stanford.edu |
| Richard Reimer | Neurotransmitter synthesis and packaging | rjreimer@stanford.edu |
| Anthony Ricci | Auditory hair cell mechanotransduction and synaptic transmissio | tricci@ohns.stanford.edu |
| Robert Sapolsky | Neuron death, stress, gene therapy | sapolsky@stanford.edu |
| Gregory Scherrer | Cellular and molecular mechanisms of pain and its control by opioids | gs25@stanford.edu |
| Matthew Scott | Cerebellum development and cancer, neural control of growth | mscott@stanford.edu |
| Carla Shatz | molecular mechanisms of experience-dependent circuit formation | cshatz@stanford.edu |
| Kang Shen | moleular mechanisms of circuit assembly at the level of synapse | kangshen@stanford.edu |
| Stephen Smith | Neural Circuit Molecular Architectures | sjsmith@stanford.edu |
| Gary Steinberg | Pathophysiology and treatment of focal and global cerebral ischemia | steinberg@stanford.edu |
| Thomas Sudhof | Formation, function, and dysfunction of synapses | tcs1@stanford.edu |
| Xinnan Wang | Mitochondria in Neuronal Health and Disease | xinnanw@stanford.edu |
| Marius Wernig | Reprogramming, induced neuronal (iN) cells, pluripotent stem cells, di | wernig@stanford.edu |
| Yanmin Yang | Cytoskeletal function and dysfunction in the nervous system | yyanmin@stanford.edu |
| David Yeomans | Pain: differential activation; gene therapy; sodium channels | dcyeomans@stanford.edu |
| Heng Zhao | stroke, postconditioning, peripheral immune system and brain injury | hzhao@stanford.edu |
-
Katrin Andreasson
-
Stephen Baccus
-
Ben Barres
-
Helen Blau
-
Kwabena Boahen
-
Anne Brunet
-
Axel Brunger
-
Paul Buckmaster
-
Marion Buckwalter
-
Lu Chen
-
Xiaoke Chen
-
Yoon-Jae Cho
-
Thomas Clandinin
-
Corrina Darian-Smith
-
Karl Deisseroth
-
Firdaus Dhabhar
-
Jun Ding
-
Ricardo Dolmetsch
-
Amit Etkin
-
Russell Fernald
-
Surya Ganguli
-
Craig Garner
-
Joseph Garner
-
Rona Giffard
-
Lisa Giocomo
-
Aaron Gitler
-
Gary Glover
-
Miriam Goodman
-
Ian Gotlib
-
Kalanit Grill-Spector
-
James Gross
-
May Han
-
H. Craig Heller
-
Shaul Hestrin
-
Ting-Ting Huang
-
John Huguenard
-
Seung Kim
-
David Kingsley
-
Eric Knudsen
-
Brian Knutson
-
Brian Kobilka
-
Ron Kopito
-
Jin Hyung Lee
-
Richard Lewis
-
Fei-Fei Li
-
Y. Joyce Liao
-
Michael Lin
-
Frank Longo
-
Bingwei Lu
-
Liqun Luo
-
David Lyons
-
Sean Mackey
-
Daniel Madison
-
Merritt Maduke
-
Robert Malenka
-
James McClelland
-
Samuel McClure
-
Susan McConnell
-
Vinod Menon
-
Michelle Monje-Deisseroth
-
Tirin Moore
-
Mirna Mustapha
-
William Newsome
-
Anthony Norcia
-
Theo Palmer
-
Karen Parker
-
Josef Parvizi
-
Giles Plant
-
Kathleen Poston
-
Thomas Rando
-
Jennifer Raymond
-
Richard Reimer
-
Allan Reiss
-
Anthony Ricci
-
Robert Sapolsky
-
Gregory Scherrer
-
Mark Schnitzer
-
Matthew Scott
-
Carla Shatz
-
Kang Shen
-
Krishna Shenoy
-
Stephen Smith
-
Gary Steinberg
-
Thomas Sudhof
-
Patrick Suppes
-
Anthony Wagner
-
Brian Wandell
-
Xinnan Wang
-
Marius Wernig
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Tony Wyss-Coray
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Yanmin Yang
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David Yeomans
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Jamie Zeitzer
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Heng Zhao
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Luis de Lecea